Diseases List

ID 141
Name CHRONIC RENAL FAILURE (CRF) (CHRONIC KIDNEY DISEASE- CKD)
Cause
Signs Symptoms
Diagnosis Diagnostic features:’ 1. Persistent & progressive uremia over weeks and months. 2. Features of many primary glomerular and tubular diseases. 3. Urinary abnormalities are usually according to the underlying diseases. 4. In majority cases heypertension develop.
Investigations Investigation: For stages 1-3 CKD: 1. Full blood count- to see anemia and other blood pictures. 2. Blood glucose & HbAlc- to know diabetes and its condition. 3. B. urea, S. creatinine & S. electrolytes- to evaluate kidney function. 4. S. albumin- low albumin; consider malnutrition, inflammation. 5. C- reactive protein- high CRP indicates sepsis or inflammatory disease. 6. Lipid profile- high in CKD, a cardiovascular risk. 7. S. calcium, S. phosphate- to assess renal osteodystrophy. 8. Parathyroid hormone- to assess renal osteodystrophy. 9. Urinalysis- to see infection, haematuria, proteinuria (indicates risk of progressive CKD requiring preventive ACE inhibitor or ARB therapy). 10.Serological tests for hepatitis and HIV. 11. Renal ultrasound- to see kidney size & exclude obstruction or progressive CKD. 12. ECG- to exclude hyperkalemia or any risk factors for cardiac disease.
Management Management: Management of stages 1-3 CKD: Most of the patients with stages 1-3 CKD, fortunately they will never develop ESRD- this is mainly due to the steep increase in the prevalence of CKD in elderly patients, particularly over the age of 70. Recommended management includes: General medical (& cardiovascular) advice: 1. Blood pressure control: Maximum target 130/80mmHg, reduced to 125/75mmHg in diabetes mellitus. Use of ACE inhibitors or angiotensin receptor blockers (ARBs) in those with proteinuria (anyone with an elevated PCR or ACR). 3. Lipid management: Dietary counselling and lipid-lowering therapies reduce risk of cardiovascular diseases. 4. Lifestyle advice: Regarding smoking, exercise, diet and weight. Nephrologist advice is required: Those patients who have potentially treatable underlying disease or who are deterioating renal function and likely to progress to ESRD should be referred to a nephrologist, such as: 1. Young age patients. 2. Renal damage referring to a nephrologist is recommended at- i. Stage 4 CKD, (in the absence of other indications), ii. Deteriorating renal function (e.g GFR fall >5ml/min/1.73m2 in 1 year, or >10ml/min/1.73m2 over 5 years)- monitoring can be reduced to annual if disease is stable or very slowly progressive, iii. Proteinuria: PCR >100mg/mmol or ACR >70mg/mmol has been suggested as a referral threshold, but this should be interpreted with reference to age comorbidity and other factors, iv. Hematuria: May be a marker for inflammatory nephritis. Management of progressive & stage 4+ CKD: The aims of management are to: 1. Findout the underlying renal disease where possible & treat it accordingly. Additional investigations may be required to specify the therapy, e.g immunosuppression in some types of glomerulonephritis. 2. Detection of reversible factors which are making renal function worse, (such as urinary tract obstruction, UTI, hypotension due to drug treatment, salt and water depletion, or nephrotoxic medication) & correct it properly. 3. Prevent further renal damage by maintaining proper diet, fluid and electrolytes. 4. Limit the adverse effects of the loss of renal function. 5. Address any associated cardiovascular risk/disease. 6. Renal replacement therapy as supportive measure (Dialysis): When progressive fall of renal functions occur. 7. Renal replacement therapy (as kidney transplantation) when appropriate.
Introduction Chronic renal failure (currently termed as- chronic kidney disease) is an irreversible deterioration in renal function which classically develops over a period of years. Initially, it manifests only biochemical abnormalities, but gradually there are loss of the excretory, metabolic and endocrine functions of the kidney, leading to the clinical symptoms and signs of renal failure, which are referred to as uremia. In this stage, renal replacement is the mast for survival. When death is likely without renal replacement therapy, it is called end-stae renal disease or failure (ESRD or ESRF) Stages of Chronic kidney disease (CKD): Stage 1- Mild CKD- Kidney damage with normal or high GFR (>90). Clinical presentation: Asymptomatic. Stage 2- Kidney damage and GFR 60-89. Clinical presentation: Asymptomatic. Stage 3A- Moderate CKD- Kidney damage and GFR 45-59. 3B- Moderate CKD-Kidney damage and GFR 30-44. Clinical presentation: Usually asymptomatic; Anaema in some patients at 3B; Most are non-progressive or progress very slowly. Stage 4- Severe CKD- Kidney damage with GFR 15-29. Clinical presentation: First symptoms often at GFR <20. Electrolyte problems likely as GFR falls. Stage 5- Kidney failure- with GFR <15 or on dialysis. Clinical presentation: Significant symptoms and complications usually present. Dialysis initiation varies but usually at GFR <10.
History
Etiology Etiology of CKD & ESRD: Common casuses of ‘chronic kidney disease’ and/or ‘end-stage renal disease’ are as following: 1. Congenital and inherited (5%)- polycystic kidney, Alport’s syndrome. 2. Renal artery stenosis (5%). 3. Hypertension (5-20%). 4. Glomerular diseases (10-20%)- IgA nephropathy (most common), Proliferative GN, Crescentic GN, Membranous GN, Mesangio-capillary G.N, Secondary GN. 5. Interstitial diseases (20-30%)- Chronic pyelonephritis, T.B, analgesic nephropathy, nephrocalcinosis. 6. Systemic inflammatory diseases (5-10%)- e.g SLE, vasculitis. 7. Obstructive uropathy- Calculus, BEP, retroperitonial fibrosis. 8. Diabetes mellitus (20-40%). 9. Unknown causes (5-20%).
Clinical Features Clinical features: May be asymptomatic; renal insufficiency may be revealed by discovery of proteinurea, anemia, hypertension & raised blood urea during routine check up. 1. General- anorexia, nausea, vomiting, weakness, lathergy, hiccup. 2. CVS- pericarditis, CCF, hypertension 3. Haemopoetic- anemia, decreased lymphocyte, abnormal bleeding. 4. Neurologic- Peripheral neuropathy CNS- seizure, hypertensive encephalopathy. 5. Musculoskeletal-Renal osteodystrophy, osteomalacia 6. Metabolic and endocrine dysfunction: glucose intolerence, sexual dysfunction, lipid abnorma-lities. 7. GIT-GIT bleeding 8. Electrolyte- hyponatraemia, hyperkalamia, met-abolic acidosis 9. Dermatologic- dry skin, itching.
Preventions
Treatment
Complications
Prognosis
Types
Classification
Observation
Pathology
1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25 26 27 28 29 30 31 32 33 34 35 36 37 38 39 40 41 42 43 44 45 46 47 48 49 50 51 52 53 54 55 56 57 58 59 60 61 62 63 64 65 66 67 68 69 70 71 72 73 74 75 76 77 78 79 80 81 82 83 84 85 86 87 88 89 90 91 92 93 94 95 96 97 98 99 100 101 102 103 104 105 106 107 108 109 110 111 112 113 114 115 116 117 118 119 120 121 122 123 124 125 126 127 128 129 130 131 132 133 134 135 136 137 138 139 140 141 142 143 144 145 146 147 148 149 150 151 152 153 154 155 156 157 158 159 160 161 162 163 164 165 166 167 168 169 170 171 172 173 174 175 176 177 178 179 180 181 182 183 184 185 186 187 188 189 190 191 192 193 194 195 196 197 198 199 200 201 202 203 204 205 206 207 208 209 210 211 212 213 214 215 216 217 218 219 220 221 222 223 224 225 226 227 228 229 230 231 232 233 234 235 236 237 238 239 240 241 242 243 244 245 246 247 248 249 250 251 252 253 254 255 256 257 258 259 260 261 262 263 264 265 266 267 268 269 270 271 272 273 274 275 276 277 278 279 280 281 282 283 284 285 286 287 288 289 290 291 292 293 294 295 296 297 298 299 300 301 302 303 304 305 306 307 308 309 310 311 312 313 314 315 316 317 318 319 320 321 322 323 324 325 326 327 328 329 330 331 332 333 334 335 336 337 338 339 340 341 342 343 344 345 346 347 348 349 350 351 352 353 354 355 356 357 358 359 360 361 362 363 364 365 366 367 368 369 370 371 372 373 374 375 376 377 378 379 380 381 382 383 384 385 386 387 388 389 390 391 392 393 394 395 396 397 398 399 400 401 402 403 404 405 406