Diseases List

ID 157
Name HODGKIN’S DISEASE
Cause
Signs Symptoms
Diagnosis
Investigations Investigations:1-2 1. Full blood count- normochromic, normocytic anemia, eosinophilia, thrombocytopenia. 2. Bone marrow- involvement is uncommon at the onset of the disease but may be found later. 3. Lymph node biopsy- diagnosis can be established by tissue biopsy. 4. Clinical staging investigations include- a. Inspection of Waldeyer’s ring b. Chest X-ray c. Liver and spleen isotope scan d. C.T scan of thorax and abdomen e. MRI- this is not more sensitive then C.T scan for detecting enlarged nodes, f. A staging laparotomy includes splenectomy, liver biopsy, multiple nodes biopsy.
Management Management: Treatment depends on the stage of the disease. There are two main modalities of treatment, radiotherapy and chemotherapy. A. Treatment of early-stage HL (Stages IA & HA): Chemotherapy with ABVD (Adriamycin i.e doxorubicin, Bleomycin, Vinblastine, Dacarbazine) rgimen 4 courses; followed by- Radiotherapy (adjunctive) to the involved lymph nodes. Treatment response should be assessed clinically and by repeated CT or PET (positron emission tomography- a newer scanning modality). Dosage: (as bellow or see the text) Adriamycin (doxorubicin)- 75mg/m2) as single dose every 3 weeks by slow i.v injection. Bleomycin- 15mg once or twice a week to a total dose of 225mg by slow i.v or in infusion. Chlorambucil- 6mg/m2 upto 10mg daily for days 1-14 orally. Decarbazine- this is now often replced by VP16 on account of its toxicity. Vinblastine- 6mg/m2 upto 10mg daily for days 1 & 8 by slow i.v injection. B. Treatment of advanced-stage HL (Stages IB, IIB, III & IV): Chemotherapy with ABVD (Adriamycin i.e doxorubicin, Bleomycin, Vinblastine, Decarbazine) rgimen 6-8 courses; followed by-assessment of response with CT or PET Radiotherapy is not recommended for advanced-stage HL. Dosage: (as above or see the text) C. Treatment ofHL resistant to chemotherapy & radiotherapy: The choice of treatment is autologous bone marrow transplantation.
Introduction Hodgkin’s lymphoma also known as Hodgkin’s disease- is characterised by progressive painless enlargement of lymphoid tissues throughout the body. The pathological hallmark of this disease is the Reed-Stemberg cell. It occurs in both sexes but more commonly in men, with bimodal age distribution, one peak around the 20s and second peak over the age 50 years
History
Etiology
Clinical Features Clinical features:123 1. Nontender (occasionaly tender), rubbery, discrete lymphadenopathy (commonly cervical). 2. The enlarged nodes compress adjacent structures & may produce dysphagia, dyspnoea, stridor, superior venacaval obstruction, (perticularly in the mediastinum) paraplegia, nerve root compression, & jaundice. 3. General features include- progressive weakness, intermittent low grade pyrexia, pruritus, weight loss, night sweating and lethergy. Staging of Hodgkin’s lymphoma:1-2-3 (Ann Arbor) Staging of lymphoma is necessary to determine the extent of disease & to determine whether localized treatment (radiotherapy) is indicated or if systemic chemotherapy is to be given. Stage I: Single lymph node region or one extralymphatic site involvement. Stage II: Two or more lymph node regions involvement on one side of the diaphragm or one lymphnode region plus one extralymphatic site on the same side of the diaphragm Stage III: Lymph node regions on both sides of diaphragm ± extralymphatic or splenic involvement. Stage IV: Diffuse involvement of one or more extralymphatic tissues (such as bone marrow or liver) involvement. In addition each stage is subclassified as- Stage A: No systemic symptoms and Stage B: Significant weight loss, fever or night sweats are present.
Preventions
Treatment
Complications
Prognosis
Types
Classification WHO Pathological classification:2 1. Nodular lymphocyte-predominant HL- 5% 2. Classical HL a. Nodular sclerosing- 70% b. Mixed cellularity- 20% c. Lymphocyte-rich- 5% d. Lymphocyte depleted- rare
Observation
Pathology
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